Jejunum & Small Intestine Surgery MCQs – NEET SS & INISS GI Surgery
Welcome to the jejunum and small intestine surgery question bank on MCQSurgery.com – This is a free page NEET SS, INISS, and GI Surgery super speciality exams.
🔥 New for 2025: Intestinal surgery MCQs now include updated NEET SS pattern questions with answer toggles and clinical case scenarios!
What You'll Find on This Page
- Carefully curated Jejunum and Small Intestine Surgery MCQs
- High-yield clinical and anatomical scenarios
- Questions aligned with Blumgart, Sabiston, and Mastery of Surgery
- NEET SS & INISS exam-based pattern
- Detailed explanations with textbook references
Key Intestinal Topics Covered:
- Crohn's disease and inflammatory bowel disorders
- Intestinal malrotation and congenital anomalies
- Pneumatosis intestinalis and its management
- Blind loop syndrome and bacterial overgrowth
- Immune mechanisms of the small intestine
- Small bowel obstruction and ischemia
Why Choose MCQSurgery for Intestinal Preparation?
Our team of experienced GI surgeons compiles questions modeled on real exam cases. All jejunum and small intestine MCQs are fact-checked and updated regularly to reflect changes in surgical guidelines and question trends.
🧠 Tip: Combine intestinal MCQs with our mock test series to improve speed and accuracy.
Start Practicing Intestinal MCQs
Click here to start jejunum and small intestine questions now with real-time scoring and instant feedback.
🎯 Want access to all intestinal MCQs and explanations?
Join Premium Surgery Course
Join Premium Surgery Course
FAQs – Jejunum & Small Intestine MCQs for NEET SS
1. Are these jejunum and small intestine MCQs based on the NEET SS GI Surgery syllabus?
Yes, all questions are mapped to the official GI Surgery syllabus and cover both core and clinical intestinal topics.
2. Do I get detailed explanations?
Premium members receive fully referenced answers with line-by-line reasoning and links to sources like Blumgart and Sabiston.
3. Can I take intestinal tests in exam mode?
Absolutely. You can attempt time-bound jejunum and small intestine MCQ mock tests in our Mock Test section.
Q1. Most common extra intestinal manifestation of Crohn's disease of small intestine
a) Ankylosing Spondylitis
b) Erythema Nodosum
c) Iritis
d) Ureteral Obstruction
Correct answer: b) Erythema Nodosum
Extra intestinal manifestations of Crohn's disease occur in 20-30% of patients.
The most common symptoms are skin lesions — Erythema nodosum and pyoderma gangrenosum.
Others include arthritis and arthralgias, uveitis and iritis, hepatitis, pericholangitis, and aphthous stomatitis.
Amyloidosis, pancreatitis, and nephrotic syndrome may also occur in these patients.
Extra intestinal manifestations of Crohn's disease occur in 20-30% of patients.
The most common symptoms are skin lesions — Erythema nodosum and pyoderma gangrenosum.
Others include arthritis and arthralgias, uveitis and iritis, hepatitis, pericholangitis, and aphthous stomatitis.
Amyloidosis, pancreatitis, and nephrotic syndrome may also occur in these patients.
Q2. Which of the following is the most common cause of death in Crohn's disease of small intestine?
a) Malignancy
b) Sepsis
c) Intestinal complications
d) Thromboembolic Phenomenon
Correct answer: c) Intestinal complications
Intestinal Complications (e.g., obstruction, perforation): 30-40%
Reference: Loftus EV Jr. "Clinical epidemiology of inflammatory bowel disease: incidence, prevalence, and environmental influences." Gastroenterology. 2004.
Infectious Complications (e.g., sepsis, abscesses): 25-30%
Reference: M'Koma AE. "Crohn's disease: A review of the disease and its management." Journal of Health Care and Research. 2015.
Malnutrition and Related Causes: 10-20%
Reference: Tisdall R, et al. "The role of nutrition in inflammatory bowel disease." Gastroenterology Clinics of North America. 2006.
Colorectal Cancer: 5-10%
Reference: Itzkowitz SH, et al. "Cancer in patients with inflammatory bowel disease." Gastroenterology. 2007.
Other Causes (e.g., thromboembolism, liver disease): 10-15%
Reference: Rungoe C, et al. "Causes of death in patients with inflammatory bowel disease: a population-based study." Inflammatory Bowel Diseases. 2013.
Intestinal Complications (e.g., obstruction, perforation): 30-40%
Reference: Loftus EV Jr. "Clinical epidemiology of inflammatory bowel disease: incidence, prevalence, and environmental influences." Gastroenterology. 2004.
Infectious Complications (e.g., sepsis, abscesses): 25-30%
Reference: M'Koma AE. "Crohn's disease: A review of the disease and its management." Journal of Health Care and Research. 2015.
Malnutrition and Related Causes: 10-20%
Reference: Tisdall R, et al. "The role of nutrition in inflammatory bowel disease." Gastroenterology Clinics of North America. 2006.
Colorectal Cancer: 5-10%
Reference: Itzkowitz SH, et al. "Cancer in patients with inflammatory bowel disease." Gastroenterology. 2007.
Other Causes (e.g., thromboembolism, liver disease): 10-15%
Reference: Rungoe C, et al. "Causes of death in patients with inflammatory bowel disease: a population-based study." Inflammatory Bowel Diseases. 2013.
Q3. Which of the following is not true about Pneumatosis intestinalis of small intestine?
a) It is seen equally in males and females
b) Most common location is subserosa in the jejunum
c) Operative procedures are required in most of the cases
d) It is associated with COPD and immunodeficiency states
Correct answer: c)
Operation is not required unless a complication such as volvulus, hemorrhage, intestinal obstruction or perforation develops.
Complications are seen in 1-3% cases.
On plain X-ray they appear as radiolucent areas in the bowel wall which have to be distinguished from luminal intestinal gas.
Reference: Sabiston 21st edition.
Operation is not required unless a complication such as volvulus, hemorrhage, intestinal obstruction or perforation develops.
Complications are seen in 1-3% cases.
On plain X-ray they appear as radiolucent areas in the bowel wall which have to be distinguished from luminal intestinal gas.
Reference: Sabiston 21st edition.
Q4. What is not true about blind loop syndrome?
a) It manifests as diarrheas, weight loss and deficiency of fat soluble vitamins.
b) Megaloblastic anemia is commonly seen
c) Surgery is almost always required to correct small bowel syndrome
d) Broad spectrum antibiotics are the treatment of choice
Correct answer: c)
Blind loop syndrome manifests by diarrhea, steatorrhea, megaloblastic anemia, weight loss, abdominal pain, and deficiencies of fat-soluble vitamins (A, D, E, and K), as well as neurologic disorders.
It is caused by bacterial overgrowth in stagnant areas of the small bowel due to stricture, stenosis, fistulas, or diverticulum.
Surgery is required in very few cases.
Surgical correction of the condition producing stagnation and blind loop syndrome produces a permanent cure and is indicated in those patients who require multiple rounds of antibiotics or continuous therapy.
Bacterial overgrowth can be diagnosed with cultures obtained through an intestinal tube or by indirect tests such as the 14C-xylose or 14C-cholylglycine breath tests. Once bacterial overgrowth and steatorrhea are confirmed, a Schilling test (57Co-labeled vitamin B12 absorption) may be performed, revealing a pattern of urinary excretion of vitamin B12 resembling pernicious anemia.
Blind loop syndrome manifests by diarrhea, steatorrhea, megaloblastic anemia, weight loss, abdominal pain, and deficiencies of fat-soluble vitamins (A, D, E, and K), as well as neurologic disorders.
It is caused by bacterial overgrowth in stagnant areas of the small bowel due to stricture, stenosis, fistulas, or diverticulum.
Surgery is required in very few cases.
Surgical correction of the condition producing stagnation and blind loop syndrome produces a permanent cure and is indicated in those patients who require multiple rounds of antibiotics or continuous therapy.
Bacterial overgrowth can be diagnosed with cultures obtained through an intestinal tube or by indirect tests such as the 14C-xylose or 14C-cholylglycine breath tests. Once bacterial overgrowth and steatorrhea are confirmed, a Schilling test (57Co-labeled vitamin B12 absorption) may be performed, revealing a pattern of urinary excretion of vitamin B12 resembling pernicious anemia.
Q5. What is not true about the immune mechanism in the small intestine?
a) Intestine contains more than 70% of IgA producing cells in the body
b) IgA acts by activating the complement pathway
c) IgA is produced by plasma cells in the lamina propria
d) Approximately 60% of the lymphoid cells are T cells
Correct answer: b)
The intestine contains more than 70% of the IgA-producing cells in the body.
IgA is produced by plasma cells in the lamina propria.
IgA does not activate complement and does not enhance cell-mediated opsonization or destruction of infectious organisms or antigens, which contrasts with other immunoglobulins.
Secretory IgA inhibits the adherence of bacteria to epithelial cells and prevents their colonization and multiplication.
Secretory IgA neutralizes bacterial toxins and viral activity and blocks absorption of antigens from the gut.
The intestine contains more than 70% of the IgA-producing cells in the body.
IgA is produced by plasma cells in the lamina propria.
IgA does not activate complement and does not enhance cell-mediated opsonization or destruction of infectious organisms or antigens, which contrasts with other immunoglobulins.
Secretory IgA inhibits the adherence of bacteria to epithelial cells and prevents their colonization and multiplication.
Secretory IgA neutralizes bacterial toxins and viral activity and blocks absorption of antigens from the gut.
Q6. What is not true about malrotation of the small intestine?
a) In incomplete rotation the rotation is arrested at 180 degrees
b) The small intestine lies on the right with caecum in the midline
c) Ladd's band extends from the right upper quadrant to the caecum
d) Hyper rotation is the most common form of intestinal malrotation
Correct answer: d)
There are several degrees of rotational abnormality.
Nonrotation is characterized by failure of counterclockwise rotation after return of the midgut to the abdominal cavity.
In incomplete rotation, the counterclockwise rotation is arrested at around 180 degrees. These are the most common forms of malrotation.
Associated with this abnormal fixation is a narrow intestinal mesentery and Ladd's bands. Ladd's bands represent the retroperitoneal attachments that normally fix the cecum and ascending colon to the posterior abdominal wall.
Because the right colon is more medial, the bands extend across the duodenum from the right upper quadrant to the cecum and ascending colon.
The duodenum assumes an anterior position and the colon lies posterior to the duodenum and the SMA.
If the counterclockwise rotation extends beyond 270 degrees, the cecum comes to rest in the left hypochondrium position. This rare form is called hyper-rotation.
There are several degrees of rotational abnormality.
Nonrotation is characterized by failure of counterclockwise rotation after return of the midgut to the abdominal cavity.
In incomplete rotation, the counterclockwise rotation is arrested at around 180 degrees. These are the most common forms of malrotation.
Associated with this abnormal fixation is a narrow intestinal mesentery and Ladd's bands. Ladd's bands represent the retroperitoneal attachments that normally fix the cecum and ascending colon to the posterior abdominal wall.
Because the right colon is more medial, the bands extend across the duodenum from the right upper quadrant to the cecum and ascending colon.
The duodenum assumes an anterior position and the colon lies posterior to the duodenum and the SMA.
If the counterclockwise rotation extends beyond 270 degrees, the cecum comes to rest in the left hypochondrium position. This rare form is called hyper-rotation.